Serveur d'exploration sur l'agrobacterium et la transgénèse

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The promoter of the pepper pathogen-induced membrane protein gene CaPIMP1 mediates environmental stress responses in plants.

Identifieur interne : 000631 ( Main/Exploration ); précédent : 000630; suivant : 000632

The promoter of the pepper pathogen-induced membrane protein gene CaPIMP1 mediates environmental stress responses in plants.

Auteurs : Jeum Kyu Hong [Corée du Sud] ; Byung Kook Hwang

Source :

RBID : pubmed:18936963

Descripteurs français

English descriptors

Abstract

The promoter of the pepper pathogen-induced membrane protein gene CaPIMP1 was analyzed by an Agrobacterium-mediated transient expression assay in tobacco leaves. Several stress-related cis-acting elements (GT-1, W-box and ABRE) are located within the CaPIMP1 promoter. In tobacco leaf tissues transiently transformed with a CaPIMP1 promoter-beta-glucuronidase (GUS) gene fusion, serially 5'-deleted CaPIMP1 promoters were differentially activated by Pseudomonas syringae pv. tabaci, ethylene, methyl jasmonate, abscisic acid, and nitric oxide. The -1,193 bp region of the CaPIMP1 gene promoter sequence exhibited full promoter activity. The -417- and -593 bp promoter regions were sufficient for GUS gene activation by ethylene and methyl jasmonate treatments, respectively. However, CaPIMP1 promoter sequences longer than -793 bp were required for promoter activation by abscisic acid and sodium nitroprusside treatments. CaPIMP1 expression was activated in pepper leaves by treatment with ethylene, methyl jasmonate, abscisic acid, beta-amino-n-butyric acid, NaCl, mechanical wounding, and low temperature, but not with salicylic acid. Overexpression of CaPIMP1 in Arabidopsis conferred hypersensitivity to mannitol, NaCl, and ABA during seed germination but not during seedling development. In contrast, transgenic plants overexpressing CaPIMP1 exhibited enhanced tolerance to oxidative stress induced by methyl viologen during germination and early seedling stages. These results suggest that CaPIMP1 expression may alter responsiveness to environmental stress, as well as to pathogen infection.

DOI: 10.1007/s00425-008-0824-z
PubMed: 18936963


Affiliations:


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Le document en format XML

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<term>Arabidopsis (drug effects)</term>
<term>Base Sequence (MeSH)</term>
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<term>Capsicum (genetics)</term>
<term>Cyclopentanes (pharmacology)</term>
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<term>Molecular Sequence Data (MeSH)</term>
<term>Osmotic Pressure (drug effects)</term>
<term>Oxylipins (pharmacology)</term>
<term>Plant Leaves (drug effects)</term>
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<term>Plant Proteins (genetics)</term>
<term>Plant Proteins (metabolism)</term>
<term>Promoter Regions, Genetic (genetics)</term>
<term>Pseudomonas syringae (drug effects)</term>
<term>Pseudomonas syringae (physiology)</term>
<term>Sequence Analysis, DNA (MeSH)</term>
<term>Signal Transduction (drug effects)</term>
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<term>Acétates (pharmacologie)</term>
<term>Adaptation physiologique (effets des médicaments et des substances chimiques)</term>
<term>Analyse de séquence d'ADN (MeSH)</term>
<term>Arabidopsis (effets des médicaments et des substances chimiques)</term>
<term>Capsicum (effets des médicaments et des substances chimiques)</term>
<term>Capsicum (génétique)</term>
<term>Cyclopentanes (pharmacologie)</term>
<term>Données de séquences moléculaires (MeSH)</term>
<term>Environnement (MeSH)</term>
<term>Feuilles de plante (effets des médicaments et des substances chimiques)</term>
<term>Feuilles de plante (génétique)</term>
<term>Gènes de plante (MeSH)</term>
<term>Oxylipines (pharmacologie)</term>
<term>Pression osmotique (effets des médicaments et des substances chimiques)</term>
<term>Protéines membranaires (génétique)</term>
<term>Protéines végétales (génétique)</term>
<term>Protéines végétales (métabolisme)</term>
<term>Pseudomonas syringae (effets des médicaments et des substances chimiques)</term>
<term>Pseudomonas syringae (physiologie)</term>
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<term>Stress physiologique (effets des médicaments et des substances chimiques)</term>
<term>Séquence nucléotidique (MeSH)</term>
<term>Tabac (effets des médicaments et des substances chimiques)</term>
<term>Tabac (microbiologie)</term>
<term>Transduction du signal (effets des médicaments et des substances chimiques)</term>
<term>Éthylènes (pharmacologie)</term>
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<term>Membrane Proteins</term>
<term>Plant Proteins</term>
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<term>Plant Proteins</term>
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<term>Abscisic Acid</term>
<term>Acetates</term>
<term>Cyclopentanes</term>
<term>Ethylenes</term>
<term>Oxylipins</term>
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<term>Pseudomonas syringae</term>
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<term>Tabac</term>
<term>Transduction du signal</term>
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<term>Capsicum</term>
<term>Plant Leaves</term>
<term>Promoter Regions, Genetic</term>
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<term>Capsicum</term>
<term>Feuilles de plante</term>
<term>Protéines membranaires</term>
<term>Protéines végétales</term>
<term>Régions promotrices (génétique)</term>
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<keywords scheme="MESH" qualifier="microbiologie" xml:lang="fr">
<term>Tabac</term>
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<term>Tobacco</term>
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<term>Protéines végétales</term>
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<div type="abstract" xml:lang="en">The promoter of the pepper pathogen-induced membrane protein gene CaPIMP1 was analyzed by an Agrobacterium-mediated transient expression assay in tobacco leaves. Several stress-related cis-acting elements (GT-1, W-box and ABRE) are located within the CaPIMP1 promoter. In tobacco leaf tissues transiently transformed with a CaPIMP1 promoter-beta-glucuronidase (GUS) gene fusion, serially 5'-deleted CaPIMP1 promoters were differentially activated by Pseudomonas syringae pv. tabaci, ethylene, methyl jasmonate, abscisic acid, and nitric oxide. The -1,193 bp region of the CaPIMP1 gene promoter sequence exhibited full promoter activity. The -417- and -593 bp promoter regions were sufficient for GUS gene activation by ethylene and methyl jasmonate treatments, respectively. However, CaPIMP1 promoter sequences longer than -793 bp were required for promoter activation by abscisic acid and sodium nitroprusside treatments. CaPIMP1 expression was activated in pepper leaves by treatment with ethylene, methyl jasmonate, abscisic acid, beta-amino-n-butyric acid, NaCl, mechanical wounding, and low temperature, but not with salicylic acid. Overexpression of CaPIMP1 in Arabidopsis conferred hypersensitivity to mannitol, NaCl, and ABA during seed germination but not during seedling development. In contrast, transgenic plants overexpressing CaPIMP1 exhibited enhanced tolerance to oxidative stress induced by methyl viologen during germination and early seedling stages. These results suggest that CaPIMP1 expression may alter responsiveness to environmental stress, as well as to pathogen infection.</div>
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<AbstractText>The promoter of the pepper pathogen-induced membrane protein gene CaPIMP1 was analyzed by an Agrobacterium-mediated transient expression assay in tobacco leaves. Several stress-related cis-acting elements (GT-1, W-box and ABRE) are located within the CaPIMP1 promoter. In tobacco leaf tissues transiently transformed with a CaPIMP1 promoter-beta-glucuronidase (GUS) gene fusion, serially 5'-deleted CaPIMP1 promoters were differentially activated by Pseudomonas syringae pv. tabaci, ethylene, methyl jasmonate, abscisic acid, and nitric oxide. The -1,193 bp region of the CaPIMP1 gene promoter sequence exhibited full promoter activity. The -417- and -593 bp promoter regions were sufficient for GUS gene activation by ethylene and methyl jasmonate treatments, respectively. However, CaPIMP1 promoter sequences longer than -793 bp were required for promoter activation by abscisic acid and sodium nitroprusside treatments. CaPIMP1 expression was activated in pepper leaves by treatment with ethylene, methyl jasmonate, abscisic acid, beta-amino-n-butyric acid, NaCl, mechanical wounding, and low temperature, but not with salicylic acid. Overexpression of CaPIMP1 in Arabidopsis conferred hypersensitivity to mannitol, NaCl, and ABA during seed germination but not during seedling development. In contrast, transgenic plants overexpressing CaPIMP1 exhibited enhanced tolerance to oxidative stress induced by methyl viologen during germination and early seedling stages. These results suggest that CaPIMP1 expression may alter responsiveness to environmental stress, as well as to pathogen infection.</AbstractText>
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